Depression Cause

POSTED by J1237 Jan 31, 2009 06:59PM: I have suffered with severe depression and anxiety for about 10 years.  I was very, extremely skeptical about the NeuroResearch formulas in treatment after having been on a myriad of antidepressant SSRI's.  I thought it was just a money making scheme and I was scared. Let me just say that I'm glad I did my research and I'm glad I tried it because it has made a WORLD OF DIFFERENCE.

diminished leading to the function being controlled and/or regulated not controlling properly causing symptoms of disease develop. Technically synaptic neurotransmitter levels prior to treatment in patients with disease due to neuron bundle damage are in the normal range for the population.  

  The Bundle Damage Theory and The Monoamine Theory are not mutually exclusive of each other. Instead these two theories can be viewed a complimentary in treatment. They address different mechanisms of action leading to neurotransmitter dysfunction and compromised electrical flow out of the post-synaptic neuron. The Monoamine Theory addresses low levels of neurotransmitters in the synapse as the etiology of impedance of electrical flow needed to regulate function and keep disease symptoms under control. Whereas, The Bundle Damage Theory addresses damage to the neuron structures (primarily post-synaptic) which impede the flow of electricity needed to regulate function and keep disease symptoms under control. With the monoamine theory and the bundle damage theory the flow of electrical energy needed to regulate function is not adequate. Differentiation of the two theories lies in the etiology of the dysfunction. Under monoamine theory returning neurotransmitter levels to normal will relieve disease symptoms. Under the bundle damage theory synaptic neurotransmitter levels need to be established that are higher than the reference range of the population.

  It is the mechanical damage to the post-synaptic neurons as suggested by the bundle damage theory and not the synaptic neurotransmitter levels that is the primary cause of monoamine disease. This subset is composed of about 88% of adult patients and 100% of the elderly patients with depressive symptoms – the nonresponders from the depression studies above.

  Neurons are intended to function for life. Loss of a neuron to apoptosis is permanent, although in limited areas of the brain neurons may regenerate to replace the neurons that have undergone apoptosis.⁵⁸ As neurons go into apoptosis in the post-synaptic neuron becomes completely non-functional they tend to go through an agonizing death where the electrical brilliance with which they function slowly fades until the electrical flow through the neuron regulating function decreases and stops over time.

  The only way to increase neurotransmitter levels in the central nervous system is to administer amino acid precursors which cross the blood brain barrier and are then synthesized into neurotransmitters. Increasing neurotransmitter levels in the synapse is analogous to increasing the voltage in an electrical wire, where by turning up the voltage you get more electricity out the other end of the wire. Turning up the voltage increases the electrical potential (pressure) of the electrons entering a partially damaged wiring connection leading to more electrons (electricity) flowing out the other end. In the case of neurotransmitter disease where the neurons of the neuron bundles are damaged to the point that the electricity flowing out of the neuron bundles is diminished disease develops. Increasing neurotransmitter levels will effectively increase voltage in the remaining viable neurons in the bundle causing electrical flow out of the damaged neuron bundles to increase to the point that normal regulation and/or control is once again observed. In this state, from a clinical standpoint, the symptoms of disease are under control.

  Its no secret, NeuroResearch Clinics uses the nutrients 5-HTP, tyrosine, levodopa, and cysteine for treatment of medical patients in order to get these results. Proper use of these simple ingredients in medical treatment is not simple. From time to time a patient will say, "Why do I want to take that, I can go to a health food store and buy it?" People off the street buying in a health food store is like going to an art store and buying a bunch of oil paints then going home and expecting to paint like a mater artist even though there was no previous painting experience. These nutrients have tremendous potential due to their chemical properties. This potential is only fully realized in the hands of the trained professional using neurotransmitter testing. Treatment is not just giving a nutrient pill, it is the whole medical management approach doctors are trained in to insure that that treatment is on track in order to get symptoms under control.

The Peer Reviewed Research of NeuroResearch Clinics

  The neurotransmitter treatment research findings of NeuroResearch Clinics have not been ignored. The University of Minnesota Medical School is writing a series of papers on the neurotransmitter testing research of NeuroResearch Clinics. The first of a series of papers by University of Minnesota Medical School based on the neurotransmitter testing research of NeuroResearch was published in "NeuroPsychiatric Disease and Treatment" May 1, 2009. Ingrid Kohlstadt, MD of Johns Hopkins in her new medical text book released February 7, 2009 included a chapter on depression written by Marty L. Hinz, MD  President Clinical Research NeuroResearch Clinics outlining in depth the proper use of amino acids and neurotransmitter testing in treatment of depression.

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