On this web site is not an alternative way to treat depression, ADHD, etc., this is the right way to treat depression, ADHD, etc. without making the cause of the problem worse.

  Overview of the most popular depression drugs in medicine are, "the reuptake inhibitor depression drugs". Make no mistake, these depression drugs deplete neurotransmitter (serotonin and dopamine) levels. Each of these depression drugs have between 15 and 35 pages of side effects listed by the depression drug companies including suicide. The depression drugs are expensive and they are only better than in a sugar pill in about 7% of patients treated for depression. The biggest problem with their use is they are habit forming (patients don't like the way they feel when they try and stop the depression drugs). The link to the right leads to an in-depth discussion a discussion of why retake inhibitor depression drugs are habit forming when used to treat depression, ADHD, etc., why their effectiveness is low, and how they deplete the neurotransmitter molecules of serotonin and dopamine.

  Neurotransmitter (serotonin and dopamine) diseases (depression, ADHD, etc.) relating to serotonin and the catecholamines (dopamine, norepinephrine, and epinephrine) are far reaching and a lengthy list. Most patients with one serotonin and/or dopamine neurotransmitter disease such as depression, ADHD, etc. actually have 3, 4, or more serotonin and/or dopamine neurotransmitter diseases active at the same time. This link is a discussion of diseases such as depression, ADHD, etc. are caused by or associated with serotonin and/or dopamine.

  Reuptake inhibitor depression drugs to nothing to increase the number of serotonin or dopamine neurotransmitter molecules in the brain. In fact reuptake inhibitor depression and ADHD drugs deplete serotonin and dopamine neurotransmitter levels. The only way to increase the serotonin and dopamine neurotransmitter levels in the brain is by giving 5-HTP aka 5HTP or 5 HTP, tryptophan, tyrosine aka L-tyrosine, or L-dopa aka dopa. Giving 5-HTP aka 5HTP or 5 HTP, tryptophan, tyrosine aka L-tyrosine, and L-dopa aka dopa must be done in proper balance or they will deplete serotonin and dopamine neurotransmitter (serotonin and dopamine) levels just as the reuptake inhibitor depression drugs do.

  Relief of symptoms in treatment of serotonin and dopamine related disease  such as depression, ADHD, etc. with 5-HTP aka 5HTP or 5 HTP, tyrosine aka L-tyrosine, and L-dopa aka dopa is like a light switch, "it is on or off". Dosing needs of 5-HTP aka 5HTP or 5 HTP, tyrosine aka L-tyrosine, and L-dopa aka dopa need to be individualized in treatment of depression, ADHD, etc., one size does not fit all. The on/off effect is so dramatic that in some patients with depression, ADHD, etc. changing the daily dose of 5-HTP aka 5HTP or 5 HTP, tyrosine aka L-tyrosine, and L-dopa aka dopa by one or two pills is the difference between relief and no relief of symptoms. Add to this the need to have the serotonin and dopamine precursors 5-HTP aka 5HTP or 5 HTP, tyrosine aka L-tyrosine, and L-dopa aka dopa in proper balance in treatment of depression, ADHD, etc. and individual dosing needs become very specific.

  Contrary to the urban legends on the internet the 5-HTP aka 5HTP or 5 HTP, tryptophan, tyrosine aka L-tyrosine, and L-dopa aka dopa are safe to use with all prescription drugs including in the treatment of depression, ADHD, etc. with depression drugs. While the side effect profile of 5-HTP aka 5HTP or 5 HTP, tyrosine aka L-tyrosine, and L-dopa aka dopa used to treat depression, ADHD, etc. when taken with no prescription drugs is similar to placebo (sugar pill) there are occasional problems that do arise. With proper management of these problems there is no patient being treated for depression, ADHD, etc. that should not be able to tolerate properly balanced 5-HTP aka 5HTP or 5 HTP, tyrosine aka L-tyrosine, and L-dopa aka dopa.

  In the 1990s prior to the work of NeuroResearch it was thought that baseline "urinary neurotransmitter testing" (serotonin and dopamine) prior to treatment was the way to go in treatment of depression, ADHD, etc. The problem doctors treating depression, ADHD, etc. under the approach of getting baseline urinary serotonin and dopamine testing could not see the forest for the trees. Under the baseline urinary serotonin and dopamine approach in treatment of depression, ADHD, etc. very few patients actually go full or any relief of symptoms. Fact is there is not correlation between giving or not giving 5-HTP aka 5HTP or 5 HTP and urinary serotonin levels. The only correlation that exists is when the phase of urinary serotonin is determined in treatment of depression, ADHD, etc. while giving 5-HTP aka 5HTP or 5 HTP. Baseline "urinary neurotransmitter testing" (serotonin and dopamine) prior to treating for depression, ADHD, etc. with 5-HTP aka 5HTP or 5 HTP, tyrosine aka L-tyrosine, and L-dopa aka dopa is a waste of time and money. Baseline urinary serotonin and dopamine neurotransmitter testing in treatment of depression, ADHD, etc. is better at lining the lab bank account with money than achieving relief of depression, ADHD, etc. symptoms.

  The comprehensive writings from the University of Minnesota Medical School papers on the ground breaking work of NeuroResearch with urinary serotonin and dopamine neurotransmitter testing. This is the testing used by NeuroResearch to treat depression, ADHD, etc.

  Neurotoxins cause brain damage to the serotonin and dopamine neurons as well as other neurons of the brain. The leading cause of chronic serotonin and dopamine neurotransmitter disease such as depression, ADHD, etc. is damage to the serotonin and dopamine neurons of brain by neurotoxins. This link is the most comprehensive list of neurotoxins we could find. While you may not recognize many of these neurotoxins which potentially cause damage to the serotonin and dopamine neurons, but you surely will recognize some. Chronic depression, ADHD, etc. is caused by neurotoxins.

  NeuroResearch is concerned that use of faba beans for its L-dopa aka dopa for treatment of depression, ADHD, etc. is gaining a resurgence. In people with glucose-6-phosphatase deficiency (G-6-P) faba beans can kill or cause severe disability. The incidence of G-6-P in certain groups is as high as 12%.

  Besides being a consultant for medical textbooks and working in conjunction with medical schools and universities studying depression, ADHD, etc. caused by serotonin and dopamine neurotransmitter levels that are not high enough, NeuroResearch Clinics, Inc. is dedicated relating up to date medical research relating to serotonin and dopamine neurotransmitter with neurotransmitter disease such as depression, ADHD, etc. to the medical profession. To meet this objective, NeuroResearch is certified to speak at formal AMA certified category I continuing medical education conferences. This caliber of teaching is so specialized that we are the only AMA certified source for continuing medical education on the topic of serotonin and dopamine neurotransmitter molecule optimization with natural 5-HTP aka 5HTP or 5 HTP, tyrosine aka L-tyrosine, and L-dopa aka dopa therapy with and without urinary serotonin and dopamine neurotransmitter  laboratory testing. Our lecturers are the licensed medical doctors who have developed the manipulation of urinary serotonin and dopamine neurotransmitter approach with 5-HTP aka 5HTP or 5 HTP, tyrosine aka L-tyrosine, and L-dopa aka dopa and continue ongoing research of serotonin and dopamine neurotransmitter disease such as depression, ADHD, etc. at present.

  Neurotransmitter (serotonin and dopamine) dysfunction disease (depression, ADHD, etc.) symptoms, such as symptoms of depression, develop when the electrical flow through the neuron bundles that regulate function is compromised by damage to the individual neurons or the neuron components composing the neuron bundle which conducts electricity to regulate or control function. In order to optimally restore neuron bundle regulatory function, synaptic neurotransmitter (serotonin and dopamine) levels of the remaining viable neurons must be increased to levels higher than is normally found in the system, which restores adequate electrical outflow resulting in relief of symptoms and optimal regulatory function.